Cancer Fighting Strategies

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Our genes have little switches on them. Most people don't get enough vitamin D from the sun, even in the south. But, it is important that if you feel a lump to speak with a medical professional. Colon and liver cleanses can be accomplished with a variety of herbs, green drinks and easily digested whole foods such as juiced vegetables or those that are lightly steamed. The researchers also found a higher rate of cancer cell death.

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They compared tumor growth in mice treated with Avemar alone, or treated with three of the most widely used and studied breast cancer drugs: Tamoxifen, Aromasin, or Arimidex, alone, and combinations of those drugs with Avemar.

Estrogen-blocking drugs are not effective against ER- breast cancers, so they were not tested in this tumor model. Chief among these mechanisms is its ability to interfere with the excess use of glucose by cancer cells the Warburg effect , which interferes with the synthesis of DNA needed for cancer cell proliferation. Avemar is not advised for pregnant women or nursing mothers. It is recommended those with cancer take 8.

To order visit [www. A patient will typically present with an emotional "pain body" self-rated at out of a possible , which is very high.

Finding a way to remove this cancer-causing stress is critical to recovery from cancer. By the end of the four 2-hour EFT sessions, the patient is normally able to rate the size of their pain body below 10 out of a possible , and frequently at zero. As revealed in the 6 phases of cancer , it is suppressed negative emotions principally anger, hate, resentment and grief which cause and continue to fuel cancer at the cellular level. Finding a way to remove these toxic emotions is critical to long term cancer recovery.

It is recommended you undertake sessions with an experienced healer of emotions such as an EFT specialist who can work with you to permanently remove these toxic emotions. The Vipassana meditation technique is also beneficial for uncovering and releasing toxic emotions.

This is because those diagnosed with cancer have significantly elevated stress hormone cortisol levels, which deplete all-important adrenaline reserves within the body in phase 2 of cancer , breaking the cell's Kreb's Citric Acid Cycle, causing cell mutation and cancer. By removing anything in your life that is causing significant stress, this will help to normalize cortisol and adrenaline levels, and thus halt the condition known as cancer which is fuelled by chronic stress.

Over many years the typical cancer personality has trained their body to remain rigid and tense in response to life stressors. And when the body is not relaxed the mind will not relax sufficiently enough to enter the deep-sleep-cycle to produce melatonin, which is the primary hormone responsible for inhibiting cancer cell growth.

You should ideally spend 2 hours each day in active relaxation mode to lower stress hormone cortisol levels, which in turn will help restore adrenaline reserves and enable you to enter the deep-sleep-cycle to produce melatonin. Here are some ways to actively relax: As revealed in phase 1 of cancer , melatonin is the primary hormone responsible for inhibiting cancer cell growth. It does this by producing interleukin 2 IL-2 which governs the production of cancer killing immune system T cells, B cells, natural killer cells, macrophages and neutrophils.

Melatonin is produced in the pineal gland of the brain between the hours of 1am and 3am in the morning during uninterrupted deep sleep. Removing the toxic emotions that disrupt deep sleep and lowering stress hormone cortisol levels will naturally correct the problem, however studies have demonstrated meditation can also be used to produce melatonin by stimulating the pineal gland.

Consider meditating for 30 minutes per day as part of your 2 hours of daily active relaxation. There are a number of things that suppress or weaken the immune system, including high stress hormone cortisol levels, depleted melatonin and dopamine levels, parasites, pathogen microbes viruses, bacteria, fungus , as well as chemotherapy and radiation.

When the immune system is suppressed or weakened, the "cancer fungus" in phase 3 thrives. We recommend you incorporate at least one protocol to support and boost your immune system.

If you are undertaking chemotherapy or radiation, consider Graviola capsules to prevent side-effects such as hair loss, nausea, and general malaise and energy loss. This natural product really works and prevents cell-resistance to chemotherapy. As revealed by the Holy Spirit of God in phase 3 of cancer , what we know as cancer is in fact seven different types of fungus.

When the cancer personality experiences prolonged chronic stress, somatids tiny microorganisms necessary for life that live in our body pleomorphise [or change] into yeast-like-fungus to ferment rising glucose and lactic acid in cells.

In a healthy person, somatids are limited to 3 stages in their life cycle - somatid, spore, double spore. However, in a highly acidic low pH lactic acid environment, somatids pleomorphise into a further 13 stages.

These stages include viral-bacterial-yeast-like-fungus forms that: For this reason it is recommended you include at least one of the following protocols to remove and keep at bay the cancer-fungus in your body: Toxins include "mycotoxins" or acidic waste products caused by: These toxins build up primarily in the liver--the master immune system organ.

When the liver is overloaded with these toxins, your immune system is weakened and you feel sicker, and cancer and viral-bacterial-yeast-like-fungus thrives. Thus it is very important to have a plan to detox the liver the master immune system organ , the colon the intestinal immune system , as well as the gall bladder and kidneys--especially if you are undertaking a treatment to kill cancer cells or the cancer fungus.

If you don't, your liver cannot remove all the dead microbes and cancer cells, which remain overloaded in the liver. Ozonated Water should be considered for it is a superb body detoxifier, but should NOT be used by those with lung cancer or lung conditions. Cancer can only exist when the Krebs' Citric Acid Cycle of a person's body cells is broken. Results from pharmacokinetic studies indicate that oral doses of 1. The total body content of vitamin C ranges from mg at near scurvy to about 2 g [ 4 ].

High levels of vitamin C millimolar concentrations are maintained in cells and tissues, and are highest in leukocytes white blood cells , eyes, adrenal glands, pituitary gland, and brain. Relatively low levels of vitamin C micromolar concentrations are found in extracellular fluids, such as plasma, red blood cells, and saliva [ 4 ]. DRI is the general term for a set of reference values used for planning and assessing nutrient intakes of healthy people.

These values, which vary by age and gender [ 8 ], include:. Table 1 lists the current RDAs for vitamin C [ 8 ]. The RDAs for vitamin C are based on its known physiological and antioxidant functions in white blood cells and are much higher than the amount required for protection from deficiency [ 4 , 8 , 11 ]. For infants from birth to 12 months, the FNB established an AI for vitamin C that is equivalent to the mean intake of vitamin C in healthy, breastfed infants.

Fruits and vegetables are the best sources of vitamin C see Table 2 [ 12 ]. Citrus fruits, tomatoes and tomato juice, and potatoes are major contributors of vitamin C to the American diet [ 8 ]. Other good food sources include red and green peppers, kiwifruit, broccoli, strawberries, Brussels sprouts, and cantaloupe see Table 2 [ 8 , 12 ].

Although vitamin C is not naturally present in grains, it is added to some fortified breakfast cereals. The vitamin C content of food may be reduced by prolonged storage and by cooking because ascorbic acid is water soluble and is destroyed by heat [ 6 , 8 ].

Steaming or microwaving may lessen cooking losses. Fortunately, many of the best food sources of vitamin C, such as fruits and vegetables, are usually consumed raw. Consuming five varied servings of fruits and vegetables a day can provide more than mg of vitamin C.

DVs were developed by the U. Food and Drug Administration FDA to help consumers compare the nutrient contents of products within the context of a total diet.

The DV for vitamin C is 60 mg for adults and children aged 4 and older. Department of Agriculture's USDA's Nutrient Database Web site lists the nutrient content of many foods and provides a comprehensive list of foods containing vitamin C arranged by nutrient content and by food name.

Supplements typically contain vitamin C in the form of ascorbic acid, which has equivalent bioavailability to that of naturally occurring ascorbic acid in foods, such as orange juice and broccoli [ ]. A few studies in humans have examined whether bioavailability differs among the various forms of vitamin C. Another study found no differences in plasma vitamin C levels or urinary excretion of vitamin C among three different vitamin C sources: These findings, coupled with the relatively low cost of ascorbic acid, led the authors to conclude that simple ascorbic acid is the preferred source of supplemental vitamin C [ 16 ].

Mean intakes for children and adolescents aged years range from Although the — NHANES analysis did not include data for breastfed infants and toddlers, breastmilk is considered an adequate source of vitamin C [ 8 , 13 ].

Use of vitamin C-containing supplements is also relatively common, adding to the total vitamin C intake from food and beverages.

Vitamin C status is typically assessed by measuring plasma vitamin C levels [ 4 , 13 ]. Other measures, such as leukocyte vitamin C concentration, could be more accurate indicators of tissue vitamin C levels, but they are more difficult to assess and the results are not always reliable [ 4 , 9 , 13 ].

Acute vitamin C deficiency leads to scurvy [ 7 , 8 , 11 ]. Initial symptoms can include fatigue probably the result of impaired carnitine biosynthesis , malaise, and inflammation of the gums [ 4 , 11 ]. As vitamin C deficiency progresses, collagen synthesis becomes impaired and connective tissues become weakened, causing petechiae, ecchymoses, purpura, joint pain, poor wound healing, hyperkeratosis, and corkscrew hairs [ 1 , 2 , 4 , ].

Additional signs of scurvy include depression as well as swollen, bleeding gums and loosening or loss of teeth due to tissue and capillary fragility [ 6 , 8 , 9 ]. Iron deficiency anemia can also occur due to increased bleeding and decreased nonheme iron absorption secondary to low vitamin C intake [ 6 , 11 ]. In children, bone disease can be present [ 6 ].

Left untreated, scurvy is fatal [ 6 , 9 ]. Until the end of the 18 th century, many sailors who ventured on long ocean voyages, with little or no vitamin C intake, contracted or died from scurvy. During the mids, Sir James Lind, a British Navy surgeon, conducted experiments and determined that eating citrus fruits or juices could cure scurvy, although scientists did not prove that ascorbic acid was the active component until [ ].

Today, vitamin C deficiency and scurvy are rare in developed countries [ 8 ]. Vitamin C deficiency is uncommon in developed countries but can still occur in people with limited food variety. The following groups are more likely than others to be at risk of obtaining insufficient amounts of vitamin C. Studies consistently show that smokers have lower plasma and leukocyte vitamin C levels than nonsmokers, due in part to increased oxidative stress [ 8 ].

For this reason, the IOM concluded that smokers need 35 mg more vitamin C per day than nonsmokers [ 8 ]. Exposure to secondhand smoke also decreases vitamin C levels. Although the IOM was unable to establish a specific vitamin C requirement for nonsmokers who are regularly exposed to secondhand smoke, these individuals should ensure that they meet the RDA for vitamin C [ 4 , 8 ].

For many reasons, feeding infants evaporated or boiled cow's milk is not recommended. This practice can cause vitamin C deficiency because cow's milk naturally has very little vitamin C and heat can destroy vitamin C [ 6 , 12 ]. Although fruits and vegetables are the best sources of vitamin C, many other foods have small amounts of this nutrient. Thus, through a varied diet, most people should be able to meet the vitamin C RDA or at least obtain enough to prevent scurvy.

People who have limited food variety—including some elderly, indigent individuals who prepare their own food; people who abuse alcohol or drugs; food faddists; people with mental illness; and, occasionally, children—might not obtain sufficient vitamin C [ 4 , , 11 ].

People with severe intestinal malabsorption or cachexia and some cancer patients might be at increased risk of vitamin C inadequacy [ 26 ]. Low vitamin C concentrations can also occur in patients with end-stage renal disease on chronic hemodialysis [ 27 ]. This section focuses on four diseases and disorders in which vitamin C might play a role: Epidemiologic evidence suggests that higher consumption of fruits and vegetables is associated with lower risk of most types of cancer, perhaps, in part, due to their high vitamin C content [ 1 , 2 ].

Vitamin C can limit the formation of carcinogens, such as nitrosamines [ 2 , 28 ], in vivo; modulate immune response [ 2 , 4 ]; and, through its antioxidant function, possibly attenuate oxidative damage that can lead to cancer [ 1 ]. Most case-control studies have found an inverse association between dietary vitamin C intake and cancers of the lung, breast, colon or rectum, stomach, oral cavity, larynx or pharynx, and esophagus [ 2 , 4 ].

Plasma concentrations of vitamin C are also lower in people with cancer than controls [ 2 ]. However, evidence from prospective cohort studies is inconsistent, possibly due to varying intakes of vitamin C among studies. Evidence from most randomized clinical trials suggests that vitamin C supplementation, usually in combination with other micronutrients, does not affect cancer risk.

MAX study, a randomized, double-blind, placebo-controlled clinical trial,13, healthy French adults received antioxidant supplementation with mg ascorbic acid, 30 mg vitamin E, 6 mg beta-carotene, mcg selenium, and 20 mg zinc, or placebo [ 32 ]. After a median follow-up time of 7.

In addition, baseline antioxidant status was related to cancer risk in men, but not in women [ 33 ]. Similar findings were reported in women participating in the Women's Antioxidant Cardiovascular Study [ 35 ]. In a large intervention trial conducted in Linxian, China, daily supplements of vitamin C mg plus molybdenum 30 mcg for 5—6 years did not significantly affect the risk of developing esophageal or gastric cancer [ 36 ].

Moreover, during 10 years of follow-up, this supplementation regimen failed to significantly affect total morbidity or mortality from esophageal, gastric, or other cancers [ 37 ]. A review of vitamin C and other antioxidant supplements for the prevention of gastrointestinal cancers found no convincing evidence that vitamin C or beta-carotene, vitamin A, or vitamin E prevents gastrointestinal cancers [ 38 ].

A similar review by Coulter and colleagues found that vitamin C supplementation, in combination with vitamin E, had no significant effect on death risk due to cancer in healthy individuals [ 39 ]. At this time, the evidence is inconsistent on whether dietary vitamin C intake affects cancer risk.

Results from most clinical trials suggest that modest vitamin C supplementation alone or with other nutrients offers no benefit in the prevention of cancer. A substantial limitation in interpreting many of these studies is that investigators did not measure vitamin C concentrations before or after supplementation. Plasma and tissue concentrations of vitamin C are tightly controlled in humans. At daily intakes of mg or higher, cells appear to be saturated and at intakes of at least mg, plasma concentrations increase only marginally [ 2 , 10 , 21 , 30 , 36 ].

If subjects' vitamin C levels were already close to saturation at study entry, supplementation would be expected to have made little or no difference on measured outcomes [ 21 , 22 , 40 , 41 ].

During the s, studies by Cameron, Campbell, and Pauling suggested that high-dose vitamin C has beneficial effects on quality of life and survival time in patients with terminal cancer [ 42 , 43 ]. However, some subsequent studies—including a randomized, double-blind, placebo-controlled clinical trial by Moertel and colleagues at the Mayo Clinic [ 44 ]—did not support these findings. The authors of a review assessing the effects of vitamin C in patients with advanced cancer concluded that vitamin C confers no significant mortality benefit [ 39 ].

Emerging research suggests that the route of vitamin C administration intravenous vs. Most intervention trials, including the one conducted by Moertel and colleagues, used only oral administration, whereas Cameron and colleagues used a combination of oral and intravenous IV administration. Concentrations of this magnitude are selectively cytotoxic to tumor cells in vitro [ 1 , 66 ].

Research in mice suggests that pharmacologic doses of IV vitamin C might show promise in treating otherwise difficult-to-treat tumors [ 48 ]. A high concentration of vitamin C may act as a pro-oxidant and generate hydrogen peroxide that has selective toxicity toward cancer cells [ ].

Based on these findings and a few case reports of patients with advanced cancers who had remarkably long survival times following administration of high-dose IV vitamin C, some researchers support reassessment of the use of high-dose IV vitamin C as a drug to treat cancer [ 3 , 46 , 48 , 51 ]. Therefore, individuals undergoing these procedures should consult with their oncologist prior to taking vitamin C or other antioxidant supplements, especially in high doses [ 53 ].

Evidence from many epidemiological studies suggests that high intakes of fruits and vegetables are associated with a reduced risk of cardiovascular disease [ 1 , 54 , 55 ]. This association might be partly attributable to the antioxidant content of these foods because oxidative damage, including oxidative modification of low-density lipoproteins, is a major cause of cardiovascular disease [ 1 , 4 , 55 ].

In addition to its antioxidant properties, vitamin C has been shown to reduce monocyte adherence to the endothelium, improve endothelium-dependent nitric oxide production and vasodilation, and reduce vascular smooth-muscle-cell apoptosis, which prevents plaque instability in atherosclerosis [ 2 , 56 ]. Results from prospective studies examining associations between vitamin C intake and cardiovascular disease risk are conflicting [ 55 ]. In the Nurses' Health Study, a year prospective study involving 85, female nurses, total intake of vitamin C from both dietary and supplemental sources was inversely associated with coronary heart disease risk [ 57 ].

However, intake of vitamin C from diet alone showed no significant associations, suggesting that vitamin C supplement users might be at lower risk of coronary heart disease. In male physicians participating in the Physicians' Health Study, use of vitamin C supplements for a mean of 5. The authors of a meta-analysis of prospective cohort studies, including 14 studies reporting on vitamin C for a median follow-up of 10 years, concluded that dietary, but not supplemental, intake of vitamin C is inversely associated with coronary heart disease risk [ 54 ].

Results from most clinical intervention trials have failed to show a beneficial effect of vitamin C supplementation on the primary or secondary prevention of cardiovascular disease.

Other clinical trials have generally examined the effects on cardiovascular disease of supplements combining vitamin C with other antioxidants, such as vitamin E and beta-carotene, making it more difficult to isolate the potential contribution of vitamin C.

The authors of a meta-analysis of randomized controlled trials concluded that antioxidant supplements vitamins C and E and beta-carotene or selenium do not affect the progression of atherosclerosis [ 65 ]. Similarly, a systematic review of vitamin C's effects on the prevention and treatment of cardiovascular disease found that vitamin C did not have favorable effects on cardiovascular disease prevention [ 66 ]. Since then, researchers have published follow-up data from the Linxian trial, a population nutrition intervention trial conducted in China [ 37 ].

Although the Linxian trial data suggest a possible benefit, overall, the findings from most intervention trials do not provide convincing evidence that vitamin C supplements provide protection against cardiovascular disease or reduce its morbidity or mortality.

However, as discussed in the cancer prevention section, clinical trial data for vitamin C are limited by the fact that plasma and tissue concentrations of vitamin C are tightly controlled in humans. AMD and cataracts are two of the leading causes of vision loss in older individuals. Oxidative stress might contribute to the etiology of both conditions. A population-based cohort study in the Netherlands found that adults aged 55 years or older who had high dietary intakes of vitamin C as well as beta-carotene, zinc, and vitamin E had a reduced risk of AMD [ 67 ].

However, most prospective studies do not support these findings [ 68 ]. The authors of a systematic review and meta-analysis of prospective cohort studies and randomized clinical trials concluded that the current evidence does not support a role for vitamin C and other antioxidants, including antioxidant supplements, in the primary prevention of early AMD [ 69 ].

Although research has not shown that antioxidants play a role in AMD development, some evidence suggests that they might help slow AMD progression [ 70 ]. After an average follow-up period of 6. A follow-up AREDS2 study confirmed the value of this and similar supplement formulations in reducing the progression of AMD over a median follow-up period of 5 years [ 72 ]. High dietary intakes of vitamin C and higher plasma ascorbate concentrations have been associated with a lower risk of cataract formation in some studies [ 2 , 4 ].

In a 5-year prospective cohort study conducted in Japan, higher dietary vitamin C intake was associated with a reduced risk of developing cataracts in a cohort of more than 30, adults aged 45—64 years [ 73 ].

Data from clinical trials are limited. In one study, Chinese adults who took daily supplements of mg vitamin C plus 30 mcg molybdenum for 5 years did not have a significantly lower cataract risk [ 75 ]. Overall, the currently available evidence does not indicate that vitamin C, taken alone or with other antioxidants, affects the risk of developing AMD, although some evidence indicates that the AREDS formulations might slow AMD progression in people at high risk of developing advanced AMD. Results of subsequent controlled studies have been inconsistent, resulting in confusion and controversy, although public interest in the subject remains high [ 79 , 80 ].

Prophylactic use of vitamin C did not significantly reduce the risk of developing a cold in the general population. When taken after the onset of cold symptoms, vitamin C did not affect cold duration or symptom severity. The use of vitamin C supplements might shorten the duration of the common cold and ameliorate symptom severity in the general population [ 79 , 82 ], possibly due to the anti-histamine effect of high-dose vitamin C [ 83 ].

However, taking vitamin C after the onset of cold symptoms does not appear to be beneficial [ 80 ]. Vitamin C has low toxicity and is not believed to cause serious adverse effects at high intakes [ 8 ].

The most common complaints are diarrhea, nausea, abdominal cramps, and other gastrointestinal disturbances due to the osmotic effect of unabsorbed vitamin C in the gastrointestinal tract [ 4 , 8 ]. The mechanism for this effect, if real, is not clear and this finding is from a subgroup of patients in an epidemiological study. No such association has been observed in any other epidemiological study, so the significance of this finding is uncertain.

High vitamin C intakes also have the potential to increase urinary oxalate and uric acid excretion, which could contribute to the formation of kidney stones, especially in individuals with renal disorders [ 8 ]. The best evidence that vitamin C contributes to kidney stone formation is in patients with pre-existing hyperoxaluria [ 22 ]. Due to the enhancement of nonheme iron absorption by vitamin C, a theoretical concern is that high vitamin C intakes might cause excess iron absorption.

In healthy individuals, this does not appear to be a concern [ 8 ]. However, in individuals with hereditary hemochromatosis, chronic consumption of high doses of vitamin C could exacerbate iron overload and result in tissue damage [ 4 , 8 ]. Under certain conditions, vitamin C can act as a pro-oxidant, potentially contributing to oxidative damage [ 8 ]. However, other studies have not shown increased oxidative damage or increased cancer risk with high intakes of vitamin C [ 8 , 89 ].

Other reported effects of high intakes of vitamin C include reduced vitamin B12 and copper levels, accelerated metabolism or excretion of ascorbic acid, erosion of dental enamel, and allergic responses [ 8 ]. However, at least some of these conclusions were a consequence of assay artifact, and additional studies have not confirmed these observations [ 8 ].

Long-term intakes of vitamin C above the UL may increase the risk of adverse health effects. The ULs do not apply to individuals receiving vitamin C for medical treatment, but such individuals should be under the care of a physician [ 8 ].

Vitamin C supplements have the potential to interact with several types of medications. A few examples are provided below. Individuals taking these medications on a regular basis should discuss their vitamin C intakes with their healthcare providers.

The safety and efficacy of the use of vitamin C and other antioxidants during cancer treatment is controversial [ 52 , 90 , 91 ].

Some data indicate that antioxidants might protect tumor cells from the action of radiation therapy and chemotherapeutic agents, such as cyclophosphamide, chlorambucil, carmustine, busulfan, thiotepa, and doxorubicin [ 53 , 90 , 92 , 93 ]. At least some of these data have been criticized because of poor study design [ 51 ].

However, due to the physiologically tight control of vitamin C, it is unclear whether oral vitamin C supplements could alter vitamin C concentrations enough to produce the suggested effects. Individuals undergoing chemotherapy or radiation should consult with their oncologist prior to taking vitamin C or other antioxidant supplements, especially in high doses [ 53 ].

It is not known whether this interaction occurs with other lipid-altering regimens [ 53 ]. Healthcare providers should monitor lipid levels in individuals taking both statins and antioxidant supplements [ 53 ]. The federal government's Dietary Guidelines for Americans notes that "Nutritional needs should be met primarily from foods.

Foods in nutrient-dense forms contain essential vitamins and minerals and also dietary fiber and other naturally occurring substances that may have positive health effects. In some cases, fortified foods and dietary supplements may be useful in providing one or more nutrients that otherwise may be consumed in less-than-recommended amounts.

For more information about building a healthy diet, refer to the Dietary Guidelines for Americans and the U. Department of Agriculture's MyPlate.

This fact sheet by the Office of Dietary Supplements ODS provides information that should not take the place of medical advice. We encourage you to talk to your healthcare providers doctor, registered dietitian, pharmacist, etc.

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